细胞培养上清 / 脐带静脉内皮细胞 / 血清 Cd137 Signaling Promotes Endothelial Apoptosis by Inhibiting Nrf2 Pathway, and Upregulating Nf-Κb Pathway 继续阅读Cd137 Signaling Promotes Endothelial Apoptosis by Inhibiting Nrf2 Pathway, and Upregulating Nf-Κb Pathway
细胞培养上清 Transient Receptor Potential Ankyrin 1 Contributes to the Atp-Elicited Oxidative Stress and Inflammation in Thp-1-Derived Macrophage 继续阅读Transient Receptor Potential Ankyrin 1 Contributes to the Atp-Elicited Oxidative Stress and Inflammation in Thp-1-Derived Macrophage
支气管肺泡灌洗液 BALF / 细胞培养上清 / 血清 Paclitaxel Alleviated Sepsis-Induced Acute Lung Injury by Activating Muc1 and Suppressing Tlr-4/Nf-Κb Pathway 继续阅读Paclitaxel Alleviated Sepsis-Induced Acute Lung Injury by Activating Muc1 and Suppressing Tlr-4/Nf-Κb Pathway
小鼠腹膜巨噬细胞 / 细胞培养上清 / 血清 USP22 suppresses the NLRP3 inflammasome by degrading NLRP3 via ATG5-dependent autophagy 继续阅读USP22 suppresses the NLRP3 inflammasome by degrading NLRP3 via ATG5-dependent autophagy
细胞培养上清 Self-Assembly of a Multifunction DNA Tetrahedron for Effective Delivery of Aptamer PL1 and Pcsk9 siRNA Potentiate Immune Checkpoint Therapy for Colorectal Cancer 继续阅读Self-Assembly of a Multifunction DNA Tetrahedron for Effective Delivery of Aptamer PL1 and Pcsk9 siRNA Potentiate Immune Checkpoint Therapy for Colorectal Cancer
RAW 264.7 / 细胞培养上清 A Network Pharmacology-Based Investigation to the Pharmacodynamic Material Basis and Mechanisms of the Anti-Inflammatory and Anti-Viral Effect of Isatis Indigotica 继续阅读A Network Pharmacology-Based Investigation to the Pharmacodynamic Material Basis and Mechanisms of the Anti-Inflammatory and Anti-Viral Effect of Isatis Indigotica
组织匀浆 / 细胞培养上清 / 结肠 Oral Core-Shell Nanoparticles Embedded in Hydrogel Microspheres for the Efficient Site-Specific Delivery of Magnolol and Enhanced Antiulcerative Colitis Therapy 继续阅读Oral Core-Shell Nanoparticles Embedded in Hydrogel Microspheres for the Efficient Site-Specific Delivery of Magnolol and Enhanced Antiulcerative Colitis Therapy
细胞培养上清 / 血清 Sars-Cov-2 N Protein Promotes Nlrp3 Inflammasome Activation to Induce Hyperinflammation 继续阅读Sars-Cov-2 N Protein Promotes Nlrp3 Inflammasome Activation to Induce Hyperinflammation
