Novus抗体推荐:细胞凋亡系列

  • Post category:生物资讯
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http://www.liankebio.com/article-information_Newsletter-846.html

作者:Novus官网 发布日期:2015-02-12 07:00

细胞凋亡(apoptosis)指为维持内环境稳定,由基因控制的细胞自主的有序的死亡。细胞凋亡与细胞坏死不同,细胞凋亡不是一件被动的过程,而是主动过程,它涉及一系列基因的激活、表达以及调控等的作用,它并不是病理条件下,自体损伤的一种现象,而是为更好地适应生存环境而主动争取的一种死亡过程。
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Caspase 3 Antibody (NB500-210)

Application: Western Blot

Sample Tested: Human Hepatoblastoma

Species: Human

Caspase 9 Antibody (NB500-209)

Application: Western Blot

Sample Tested: COLO-205 Cell Line

Species: Human
产品引用文献:
Bcl2 Antibody NBP1-72213] Am J Clin Oncol. 2008 Dec;31(6):585-8. Statistical interpretation of CA125 and Bcl-2 in serum of patients with late stage ovarian cancer. Camlica H, Duranyildiz D, Tas F, Yasasever V. Department of Preventive Oncology, Biostatistics and Epidemiology, Institute of Oncology, University of Istanbul, Istanbul, Turkey.

[Caspase 3 Antibody NB500-210] Dash R, Azab B, Quinn BA, et al. Apogossypol derivative BI-97C1 (Sabutoclax) targeting Mcl-1 sensitizes prostate cancer cells tomda-7/IL-24-mediated toxicity. Proc Natl Acad Sci U S A. 2011 May 24;108(21):8785-90.[PMID 21555592] [Caspase 3 Antibody NB500-210] Rothberg BEG, Bracken MB, Rimm DL. Tissue Biomarkers for Prognosis in Cutaneous Melanoma: A Systematic Review Meta-analysis. J Natl Cancer Inst 2009;101(7):452-474 [pubmed: 19318635] [Caspase 3 Antibody NB500-210] Kulkarni AA, Kingsbury SR, Tudzarova S, et al. Cdc7 Kinase Is a Predictor of Survival a Novel Therapeutic Target in Epithelial Ovarian Carcinoma. Clin Cancer Res 2009;15(7):2417-2425 [pubmed: 19318489] [Caspase 7 Antibody NB110-55664] Nikiforov MA, Riblett M, Tang W-H, et al. Tumor cell-selective regulation of NOXA by c-MYC in response to proteasome inhibition. PNAS. 2007 December 4, 2007;104(49):19488-93.

[Survivin Antibody NB500-201] El Ghamrasni S, Pamidi A, Halaby MJ, et al. Inactivation of chk2 and mus81 leads to impaired lymphocytes development, reduced genomic instability, and suppression of cancer. PLoS Genet.PLoS Genet. 2011 May;7(5):e1001385 [PMID 21625617]联科生物代理截止时间:2017年6月30日